Journal Articles

Permanent URI for this collectionhttps://mro.massey.ac.nz/handle/10179/7915

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Start date: 2020Subject: search.filters.subject.viral oncogenesis

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    First report of a papillomavirus-induced viral plaque in the mouth of a dog
    (John Wiley and Sons Ltd on behalf of ESVD and ACVD, 2025-05-15) Munday JS; Hobson P; Bell CM
    Canis familiaris papillomavirus type 16 was amplified from a mass in the mouth of a dog. The mass was histologically consistent with a pigmented viral plaque. This is the first report of an oral viral plaque in a dog. Histological investigation is essential to allow differentiation from an oral melanoma.
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    Amplification of Ovis aries papillomavirus type 2 DNA from an ovine cutaneous fibropapilloma.
    (John Wiley and Sons, Inc., 2024-04-01) Munday JS; Klobukowska HJ; Nicholson K
    Seven of 60 Perendale sheep within a flock developed single or multiple exophytic masses on their distal hind limbs. A mass was excised from one sheep and histological evaluation revealed epidermal and mesenchymal proliferation, papillomavirus-induced keratinocyte changes and marked keratohyalin clumping. Ovis aries papillomavirus type 2 DNA sequences were amplified using PCR.
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    Detection of a novel papillomaviral sequence in viral plaques confined to the pinna of a dog.
    (John Wiley and Sons, Inc., 2023-08-01) Munday JS; Orbell G; Robinson L
    A raised plaque that contained histological evidence of papillomavirus infection and sequences from a novel papillomavirus type developed close to the ear canal of a 14-year-old West Highland white terrier. The plaque was excised, and further plaques developed within the same area of pinna.
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    Papillomaviruses in Domestic Cats
    (MDPI (Basel, Switzerland), 2021-08-22) Munday JS; Thomson NA; Beatty JA; Tasker S
    Papillomaviruses (PVs) are well established to cause hyperplastic papillomas (warts) in humans and animals. In addition, due to their ability to alter cell regulation, PVs are also recognized to cause approximately 5% of human cancers and these viruses have been associated with neoplasia in a number of animal species. In contrast to other domestic species, cats have traditionally been thought to less frequently develop disease due to PV infection. However, in the last 15 years, the number of viruses and the different lesions associated with PVs in cats have greatly expanded. In this review, the PV life cycle and the subsequent immune response is briefly discussed along with methods used to investigate a PV etiology of a lesion. The seven PV types that are currently known to infect cats are reviewed. The lesions that have been associated with PV infections in cats are then discussed and the review finishes with a brief discussion on the use of vaccines to prevent PV-induced disease in domestic cats.